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Post-cardiac injury syndrome (PCIS) includes post-myocardial infarction syndrome and post-pericardiotomy syndrome. PCIS is usually observed 1~6 weeks after cardiac surgery or myocardial infarction, and rarely after pacemaker implantation, coronary perforation, pulmonary thromboembolism, or radio-frequency ablation. PCIS is characterized by a low-grade fever, pleuritic chest pain, myalgia, a pericardial friction rub, increased inflammatory markers, and pericardial and pleural effusions. Although the pathophysiology of PCIS is controversial, the presence of anti-heart antibodies has implicated an autoimmune response, which has been widely accepted. The treatment of PCIS includes nonsteroidal anti-inflammatory drugs and corticosteroids. Currently, intervention is being performed increasingly in complicated obstructive coronary artery disease, such as chronic total occlusion, long-segment obstructive lesion, and left main coronary artery disease. We report a very rare case of PCIS following coronary artery perforation during PCI.
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